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Dr Khan is an Assistant Professor in the Department of Neurology at University of Tennessee Health Science Centre, Memphis USA. Over the past 10 years, Dr Khan is working on regulation and resolution of neuroinflammation and neurodegeneration in experimental models of neurodegenerative diseases. His research investigates the molecular mechanisms that regulate inflammation in the central nervous system that are critical to our understanding of a broad spectrum of neurodegenerative diseases, including Parkinson’s disease, Stroke, Alzheimer’s disease, multiple sclerosis and Dystonia and to discovering new treatments for these incurable diseases. Dr Khan has published more than 45 research articles in preclinical CNS models. His research work has been recognized both at national and international levels and has received over 2400 citation scores with h-index 29 by other scientists. Dr Khan’s works has been highlighted by Editorial commentary and captured by several electronic media. One of his research article has been featured as Most Cited Brain Research article. Dr Khan been served as Academic Editor for several manuscripts submitted for the peer review process. He is an active member for several professional societies related to neuroscience and officially review the manuscripts, case reports, and Data sheet for several journals of high impact and international repute. He has been enlisted as Lead editor and editorial board member of many journals in his field and has been selected as International reviewer for National Research Foundation, South Africa. Dr Khan’s long-term research is committed to unravelling the mysteries of neurodegenerative diseases and to discovering new treatments for these incurable diseases.
Neurodegeneration and neuroinflammation in brain injuries and diseases, Alzheimer disease and AD-related Dementias, Stroke, Neuronal Stem Cells, Oxidative stress in neuropathogenesis, Neurobehavioral alterations in neurodegeneration, Antioxidants and neuroprotection, Apoptosis, Neuroinflammation in multiple sclerosis and stroke, Neuroinflammatory mediators in CNS, Stress-activated protein kinases in inflammatory signal transduction, Endothelial dysfunction, BBB disruption and immune cell mediated injury in sub- acute/ chronic phase of stroke, Brain lipids and their metabolism in neurodegeneration, Vascular cognitive impairment